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Describe the mechanism of acetaminophen hepatoxicity?
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- Describe the precipitants of ketoacidosis in this diabetic patient. What is the cause of his altered mental status?Explain Diabetic ketoacidosis?Which of the following is a clinical manifestation consistent with diabetic ketoacidosis? Question 63 options: a) Glucose < 14 mmol/L b) pH > 7.51 c) Bicarbonate > 30 mmol/L d) Acetone breath
- what class of hypertensive drugs can cause hyperglycemia for a non-diabetic patient?The pathophysiology of chronic diabetic complications is related to which process? Question 65 options: a) Oxidative stress and ROS have a limited role in the pathophysiology of diabetes mellitus b) Sorbitol and fructose are products of the polyol pathway that accumulate and reduce intracellular osmotic pressure causing cellular injury c) AGEs are formed by the irreversible binding of glucose to proteins, lipids and nucleic acids d) The hexosamine pathway causes pathologic changes in gene expression associated with increased insulin activity and improved cardiovascular healthCan the intake of alanine, glycine, and serine relieve hypoglycemia caused by starvation? Explain.
- Explain the reason(s) for considering ketoacidosis as a serious life-threatening complication in patients with diabetes mellitus.How can an individual prevent himself from having Diabetic Ketoacidosis?Which of the following best describes the role of the hexosamine pathway in the pathogenesis of the chronic complications of diabetes mellitus? Question 79 options: a) It involves irreversible binding of glucose to proteins, lipids and nucleic acids which damages components of the microcirculation leading to retinopathy, etc. b) It promotes the O-linked glycosylation of proteins and transcription factors, resulting in altered gene expression contributing to insulin resistance & cardiovascular complications c) It promotes the synthesis of DAG which increases pro-inflammatory gene expression and endothelial ET-1 production resulting in blood flow abnormalities d) It leads to intracellular accumulation of osmotically active sorbitol and fructose which damages Schwann cells, erythrocytes and the lens of the eye
- What is diabetic ketoacidosis and how does it happen?Why is amyloid toxic to cellsand how does it contribute toneurodegenerative diseases such asAlzheimer’s disease?A client with Type 1 diabetes mellitus is admitted to the hospital in a semi-conscious state with fever, nausea, and vomiting. The urine sample was positive for ketone bodies. Which of the following statements regarding the client is correct? Question 80 options: a) Glucagon should be administered to stimulate glycogenolysis and gluconeogenesis in the liver b) An injection of insulin will decrease ketone-body production c) The client should be started on a glucose infusion to help them regain consciousness d) The client's blood glucose level is well below 4.4 mmol/L