Use each of the following species concepts to write a claim about whether the dark and light fur mice are, in fact, different species. Your claim should demonstrate your understanding of that particular species concept.Mice with light coat colors are found in populations scattered across the southeastern US. Researchers believe the light coat color emerged in a single population. How would a biologist explain the evolution of the light coat color in populations across the southeastern US? Briefly explain your reasoning. Standard 6a9. Use each of the following species concepts to write a claim about whether the dark and lightfur mice are, in fact, different species. Your claim should demonstrate your understanding ofthat particular species concept.Species concept ClaimMorphologicalBiologicalEcologicalMice with light coat colors are found in populations scattered across the southeastern US.Researchers believe the light coat color emerged in a single population.10. How would a biologist explain the evolution of the light coat color in populations across thesoutheastern US? Briefly explain your reasoning. The beach mouse (Peromyscus polionotus) is a small rodent found in the southeastern UnitedStates. Coat color in the beach mouse is variable, from dark brown to blonde (Figure 1). Apigment called eumelanin is responsible for some of the diversity in mouse coat color. TheMC1R gene, located on chromosome 8, plays a major role in the synthesis of eumelanin. Thereare two alleles of MC1R:- R allele. Results in a functional MC1R protein that leads to stimulation of pigmentproduction. This allele is found in dark colored mice.C allele. Results in a non-functional MC1R protein. Pigment production is, as a result, notstimulated. This allele is found in light colored mice.Figure 1. Beach mouse with light fur (left) and dark (right).These mice are hunted by visual predators like owls and fox. The distribution of coat color is notrandom. Beach mice with dark fur are found further inland, on dark brown soils while mice withlight fur are found on beaches, where the sand is light brown. Dark fur mice also have toBiol 151 | Su 2022Page 5 of 9Problem Set 3compete with another mouse species (Peromyscus maniculatus) that inhabits a similarecological niche.

Species are defined as groups of organisms that are similar to each other and are capable of…

9. Use each of the following species concepts to write a claim about whether the dark and light fur mice are, in fact, different species. Your claim should demonstrate your understanding of that particular species concept. Species concept Morphological Biological Ecological Claim Mice with light coat colors are found in populations scattered across the southeastern US. Researchers believe the light coat color emerged in a single population. 10. How would a biologist explain the evolution of the light coat color in populations across the southeastern US? Briefly explain your reasoning.

9. Use each of the following species concepts to write a claim about whether the dark and light fur mice are, in fact, different species. Your claim should demonstrate your understanding of that particular species concept. Species concept Morphological Biological Ecological Claim Mice with light coat colors are found in populations scattered across the southeastern US. Researchers believe the light coat color emerged in a single population. 10. How would a biologist explain the evolution of the light coat color in populations across the southeastern US? Briefly explain your reasoning.

Biology: The Unity and Diversity of Life (MindTap Course List)

14th Edition

ISBN:9781305073951

Author:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Chapter26: Human Evolution

Section: Chapter Questions

Problem 1DAA: Neanderthal Hair Color The MC1R gene regulates pigmentation in humans (Sections 14.1 and 15.1...

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The beach mouse (Peromyscus polionotus) is a small rodent found in the southeastern United States. Coat color in the beach mouse is variable, from dark brown to blonde (Figure 1). A pigment called eumelanin is responsible for some of the diversity in mouse coat color. The MC1R gene, located on chromosome 8, plays a major role in the synthesis of eumelanin. There are two alleles of MC1R: R allele. Results in a functional MC1R protein that leads to stimulation of pigment production. This allele is found in dark colored mice. C allele. Results in a non-functional MC1R protein. Pigment production is, as a result, not stimulated. This allele is found in light colored mice. Figure 1. Beach mouse with light fur (left) and dark (right). These mice are hunted by visual predators like owls and fox. The distribution of coat color is not random. Beach mice with dark fur are found further inland, on dark brown soils while mice with light fur are found on beaches, where the sand is light brown.…
An STR on chromosome 5 is very closely linked to a gene involved in a dominant metabolism disorder (one one copy of the mutation causes the disease). There are four different alleles of the STR (Q, B, S, and L), each with different numbers of GATCTCG repeats. The S allele has 2 repeats, the B allele has 7 repeats, the Q allele has 10 repeats, and the L allele has 5 repeats. Part 1 Lane 1 of the gel shown below indicates the locations of PCR products corresponding to all four STR alleles when run together in one lane. DNA was loaded into the wells at the top of the gel. Move the letter of each allele to its correct position at the left of Lane 1 to indicate where each allele runs on the gel. 0000 Gabriel Andre Lane 1 O Maria Andre Maria Juliana B L Part 2 Andre and Maria are the parents in this family; all others shown on the gel are children. Daughter Juliana is born with the metabolism disorder, but daughter Camila does not have the disorder. Assuming no recombination between the STR…
It is also note that Chimpanzee has a chromosomal complement of 2N = 48, whereas human has 2N = 46. Interestingly, chromosomal banding patterns have revealed that the chromosome 12 and 13 of chimp show homology with human chromosome 2 (see figure for the alignment of human and chimp banding patterns). This is a good example of chromosomal aberration in evolution. What type of chromosomal rearrangement is likely explained the evolution of human chromosome 2 from the two chromosomes of chimpanzee? A. Translocation B. Duplication C. Inversion D. Deletion
Let’s suppose an X-linked gene in mice exists as two alleles, whichwe will call B and b. X-chromosome inactivation, a process inwhich one X chromosome is turned off, occurs in the somatic cellsof female mammals . Allele B encodes an mRNA that is 900 nucleotides long, whereas allele b contains a small deletion that shortens the mRNA to a length of 825 nucleotides.Draw the expected Northern blot that will be obtained usingmRNA isolated from somatic tissue of the followingmice:Lane 1. mRNA from an XbY male mouseLane 2. mRNA from an XbXb female mouseLane 3. mRNA from an XBXb female mouse.Note: The sample taken from the female mouse is not from aclone of cells. It is from a tissue sample, like the one shown.
The chromosome 9/22 translocation associated withCML (chronic myelogenous leukemia) is called thePhiladelphia chromosome after the city in which itscancer association was first discovered in 1960.People with CML do not inherit this translocation—it occurs in somatic cells. Why do you think that thisparticular translocation that fuses the bcr and ablgenes happens independently in the somatic cellsof many different people?
Neanderthal Hair Color The MC1R gene regulates pigmentation in humans (Sections 14.1 and 15.1 revisited), so loss-of-function mutations in this gene affect hair and skin color. A person with two mutated alleles for this gene makes more of the reddish melanin than the brownish melanin, resulting in red hair and pale skin. DNA extracted from two Neanderthal fossils contains a mutated MC1R allele that has not yet been found in humans. To see how the Neanderthal mutation affects the function of the MC1R gene. Carles Lalueza-Fox and her team introduced the allele into cultured monkey cells (FIGURE 26.16). FIGURE 26.16 MC1R activity in monkey cells transgenic for an unmutated MC1R gene, the Neanderthal MC1R allele, or the gene for green fluorescent protein (GFP). GFP is not related to MC1R. 1. How did MCR1 activity in monkey cells with the mutant allele differ from that in cells with the normal allele?
Familial retinoblastoma, a rare autosomal dominant defect, arose in a large family that had no prior history of the disease. Consider the following pedigree (the darkly colored symbols represent affected individuals): a. Circle the individual(s) in which the mutation most likely occurred. b. Is the person who is the source of the mutation affected by retinoblastoma? Justify your answer. c. Assuming that the mutant allele is fully penetrant, what is the chance that an affected individual will have an affected child?
The gene controlling ABO blood type and the gene underlying nail-patella syndrome are said to show linkage. What does that mean in terms of their relative locations in the genome? What does it mean in terms of how the two traits are inherited with respect to each other?
The dominant condition elliptocytosis causes red blood cells to become misshapen into oval-shaped cells. One of the genes responsible for the abnormal shape encodes the band 4.1 protein that together with ankyrin and other scaffold proteins creates and maintains the spherical concave shape of a normal red blood cell. The gene for band 4.1 protein, EPB41, is found on the p arm of chromosome 1. This is very close to the gene encoding the red blood cell Rhesus (Rh) blood type, either phenotype + (dominant) or - (recessive), with a recombination frequency of 2%. This means that 98% of the time alleles for these two genes are linked and are transmitted together. Diane and Jack are siblings, and both have elliptocytosis and Rh+ blood type. Due to the elliptocytosis, both had emergency splenectomies after having severe anemia. Their younger brother, Devonté, has not yet shown signs of elliptocytosis, but has Rh- blood. André, their dad, also has elliptocytosis and Rh+ blood; while their…
WHIM syndrome is a disease of the immunesystem resulting in warts and frequent infections.The disease is caused by a dominant gain-of-functionmutation in a gene on chromosome 2 called CXCR4.A 38-year-old woman suffering with WHIM syndrome her entire life was suddenly and mysteriouslycured. Genome analysis of her blood precursor cells(stem cells) revealed that many of these cells had achromosome 2 that had undergone chromotripsis—arare (and poorly understood) process where a chromosome is “shattered” into small pieces that are subsequently stitched back together in random order, resultingin many deletions and inversions. Explain howchromotripsis of chromosome 2 in a blood stem cellcould have cured the woman of WHIM syndrome.
In McCune-Albright syndrome, fibrous connective tissue replaces bone, tan patches (café-au-lait spots) dot the skin, and hormone abnormalities cause early puberty and malfunction of the thyroid, pituitary, and adrenal glands. The phenotype is highly variable, and all patients are somatic mosaics for the mutation, which is in the gene GNAS1. Why is the condition seen only in mosaics?
The Drosophila gene Sex lethal (Sxl) is deserving of itsname. Certain alleles have no effect on XY animals butcause XX animals to die early in development. Other alleles have no effect on XX animals but cause XY animals to die early in development. Thus, some Sxl allelesare lethal to females, while others are lethal to males.a. Would you expect a null mutation in Sxl to causelethality in males or in females? b. Why do Sxl alleles of either type cause lethality ina specific sex?The gene transformer (tra) gets its name from sexualtransformation, as some tra alleles can change XXanimals into morphological males, while other traalleles can change XY animals into morphologicalfemales.c. Which of these sex transformations would becaused by null alleles of tra and which would becaused by constitutively active alleles of tra?d. In contrast with Sxl, null tra mutations do notcause lethality either in XX or in XY animals.However, the Sxl protein regulates the productionof the Tra protein. Why…