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we read that in tumor cells Rb protein is hyperphosphorylated. In response to that, will p53 level increase or decrease? Why or why not?
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- L A Moving to another question will save this response. Question 5 Select the correct statements 0 An oncogene is a cancer producing gene A proto-oncogene is only found in cancer cells Tumor suppressor genes are genes whose normal products inhibit cell division A proto-oncogene is a normal cellular gene that has the potential to become an oncogene A Moving to another question will save this response.p53 is a gene / protein often associated with cancer. Why? What does p53 do? What kind of gene is it? Is it associated more with any one particular type of cancer or all cancers? Tell me more about p53, but please do not exceed one typed page.My question is two fold. What are some diseases that cause and/or are caused by apoptosis? Also if apoptosis is the most common form of programmed cell death in animals, what other forms of programmed cell death are there? Thank you.
- Determine if the gene on the left is a proto-oncogene or tumor suppressor. APC p53 Ras [Choose] [Choose] Tumor suppressor Proto-oncogene [Choose]Blackboard + > x U O O HELP CENTER Question 5 The p53 gene product (the protein made from this gene) is involved in: A the M checkpoint B) bringing cells out of GO c the length of time a cell spends in interphase D marking cells for programmed cell death Question 6 Which of the following is a reasonable estimate of the percentage of time spent in mitosis in a typical human cell 98% Scanned with CamScanner O HELP CENTER D marking cells for programmed cell death Question 6 Which of the following is a reasonable estimate of the percentage of time spent in mitosis in a typical human cell cycle? 98% B 50% 25% D 5% Question 7 If Hershey and Chase found only 355 in the bacterial cells, they would conclude that Scanned with CamScanner O HELP CENTER Question 7 If Hershey and Chase found only 3s in the bacterial cells, they would conclude that A RNA is the genetic material B) DNA and protein together serve as the genetic material C DNA is the genetic material D protein is the genetic materialYou are in charge of a new gene therapy clinic. Two cases have been referred to you for review and possible therapy. Case 1. A mutation in the promoter of a proto-oncogene causes the gene to make too much of its normal product, a receptor protein that promotes cell division. The uncontrolled cell division has caused cancer. Case 2. A mutation in an exon of a tumor-suppressor gene makes this gene nonfunctional. The product of this gene normally suppresses cell division. The mutant gene cannot suppress cell division, and this has led to cancer. What treatment options can you suggest for each case?
- "In the cellular regulatory pathways that control cell growth and proliferation, the products of oncogenes are stimulatory components and the products of tumor suppressor genes are inhibitory components" is true or false.In tumor cells Rb protein is hyperphosphorylated. In response to that, will p53 level increase or decrease? Why or why not? new answer pleaseWhat is the rationale for synthesizing and rapidly degrading p53 protein in the cell? (new answer)
- If you were to compare stem cells to an everyday object or situation, what would they be like and why? I need some examples for this. It's very hard to think of one.D Question 18 Your patient's biopsy report indicates they have "cancer in situ". This means: Abnormal cells are present, they have not invaded local cells O Cells are normal, they will become cancer cells in the near future Abnormal cells are present, they have invaded a small number of local cells Cells are normal, they will not become cancer cells unless stimulated « PreviousI am stuck on a question about cell growth signaling and RTKs. If phosphatase is nonfunctional, could it lead to uncontrollable growth resulting in cancer?