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- Can you please pick the right answer in each parentheses? Cholesterol levels in human serum are determined by ["only biosynthetic rates", "a combination of endogenous synthesis, update from the diet, and excretion via bile"] . Inhibition of ["HMG-CoA synthase", "HMG-CoA reductase"] can reduce cholesterol levels, and reduces the formation of a 6 carbon organic acid, ["isopentyl diphosphate", "mevalonate"], that is a critical building block for cholesterol.The large amount of energy used during aerobic exercise(e.g., running) requires large amounts of oxaloacetate.Explain why acetyl-CoA cannot be used to produceoxaloacetate in this circumstance. What is the sourceof oxaloacetate molecules during aerobic activity?Read these notes and question carefully! For questions on this page use these structures, they are labelled 1-4 for use in questions. These structures are intermediates in the B-oxidation pathway, not all the intermediates are shown! Remember the complete B-oxidation pathway! R = hydrocarbon chain. The acyl CoA molecule represents the first acyl-CoA in the pathway and not the last. سلسل 1 o R. 2 -CoA R. COA محمد 3 What is the correct order of the intermediates as they appear in the B-oxidation pathway? I COA 4 loca COA H3C
- CHOOSE THE CORRECT LETTER 1.What are the B-oxidation products of lauric acid (C12H2402), a major component of coconut oil?A.6 acetyl-CoA, 6 NADH, 6 FADH2B. 6 acetyl-CoA, 5 NADH, 5 FADH2C. 5 acetyl-CoA, 6 NADH, 6 FADH2D. 5 acetyl-CoA, 5 NADH, 5 FADH2 2.What is the mechanism of ATP synthesis in glycolysis?A. substrate level phosphorylationB. reductionC. oxidationD. oxidative phosphorylation 3.Which of the following enzymes need ATP as a substrate in the reaction?A.phosphoglycerate kinaseB. phosphofructokinaseC. pyruvate kinaseD.glyceraldehyde-3-phosphate dehydrogenase. Glucagon secretion causes inhibition of intracellular acetyl-CoA car- boxylase activity by several mechanisms. Name all you can think of.There are eighteen (18) rows of boxes, for around 36-38 boxes total. Please see images attached. This is a pathway trace, with the final box showing "ribose 5 phosphate" as the end product. Any help is appreciated-thank you! glutamate to ribose 5 phosphate No a ketoglutarate dehydrogenase No pyruvate carboxylase No transketolase No transaminase You do get to have if needed: erythrose 4 phosphate asparate dihydroxyacetone phosphate bicarbonate ATP ornithine CO2
- Glucagon secretion inhibits intracellular acetyl-CoA carboxylase activity through a number of different methods. Make a list of everything that comes to mind.I don't get it at all. I struggled with my homework. Can you help me? Can you help me to explain to me to get the difference between ketogenesis and ketoacidosis., please?Consider the docosanoic acid, C21H43CO2H a. Label the a and B carbons b. Draw the acyl CoA derived from this fatty acid c. How many acetyl CoA molecules are formed by complete B-oxidation? d. How many cycles of B-oxidation are needed for complete oxidation? e. How many molecules of ATP are formed from the complete catabolism of this fatty acid?
- The large amount of energy used during aerobic exercise (e.g., running) requires large amounts of oxaloacetate. Explain why acetyl-CoA cannot be used to produce oxaloacetate in this circumstance. What is the likely source of oxaloacetate molecules during aerobic activity?Below are some statements about the TCA Cycle. Which statement is true based on what we have learned in the class? Question 16 options: Oxaloacetate can be replenished from pyruvate carboxylation catalyzed by pyruvate carboxylase. The catabolism of proteins can only feed into the TCA cycle at one point, which is Acetyl-CoA. Acetyl-CoA has transporter proteins and can be transported directly from matrix to cytoplasm (for fatty acid synthesis). In TCA cycle, both Acetyl-CoA and Succinyl-CoA hydrolysis reactions are coupled with ATP/GTP synthesis.Fill in the blanks below (input numbers only!) about the metabolism of hexanoic acid, a fatty acid that is one of the components of vanilla, and whose formula is CH3(CH2)4COOH: First, the fatty acid is activated by attaching CoA, which costs ATP molecules. The fatty acid is then broken down through a beta-oxidation spiral, to make acetyl CoA molecules. This will require "turns" of the beta-oxidation process. Since each turn of the cycle yields ATP molecules, and each acetyl COA will yield ATP molecules by going through the rest of its metabolism, the net ATP molecules produced from one molecule of this fatty acid will be