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- Explain the difference between slow and fast chemical transmission. Fig. 8.23
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- Watch this video (http://openstaxcollege.org/l/summation) to learn about summation. The process of converting electrical signals to chemical signals and back requires subtle changes that can result in transient increases or decreases in membrane voltage. To cause a lasting change in the target cell, multiple signals are usually added together, or summated. Does spatial summation have to happen all at once, or can the separate signals arrive on the postsynaptic neuron at slightly different times? Explain your answer.Actions of neuropeptides include all the following, except :-a- inhibition of gene transcriptionb- decreased cyclic AMP synthesisc- changing intracellular Ca ++ leveld- activation of ligand-gated receptorsIdentifying Neurotransmitters 3) Produces response in postsynaptic cell - Qualifying condition: Molecules evoking same response as neurotransmitters Presynaptic terminal Postsynaptic. dendrite Drug-containing micropipette (753) Apply drug by passing electrical current Stimulate axon Record V Drug application through the micro-pipette Stimulated neurons Change in membrane potential AVm Outcome 1 Outcome 2 Outcome 3 0 -30 mV 15 mV 0 -15 mV -15 mV What is the interpretation of the above results?
- Absolute refractory periods in axons: Result from conformational changes in voltage gated K channels after opening O Result from Cl channel induced hyperpolarization O Result from Ca dependent phosphorylation of K channels Result from conformation changes in voltage gated Na channels after opening O Result from Ca dependent phosphorylation of Na channelsA loss-of-function mutation in the ion channel responsible for thegeneration of end-plate potentials results in a diminished permeabilityof the ion channel. 1) How would you experimentally test the effect of the mutation on thefunctional properties of the cell (synapse)? You should discuss the typesof postsynaptic responses and physiological parameters you couldmeasure.Nerve transmission and communication with other neurons. DI it restores the membrane potential the chemical that talks between one neuron and the other neuron the point between the neuron and the muscle transmits impulse to dendrite it carries receptors on its surface it produces the neurotransmitter 1. Neurotransmitter 2. Presynaptic membrane 3. Postsynaptic membrane 4. Nat-K+ pump 5. Neuromuscular junction 6. Axon
- Membrane permeability at the rising phase of an action potential is dominated by: O Co-transporters Nongated Channels O K+ Voltage-gated Channels O Na+ voltage-gated Channels O Chemically-gated ChannelsIf a neurotransmitter molecule binds to postsynaptic receptors, the result can be the resting membrane decreasing from -70 mV to -63 Mv (for example). The neuron is now conducted hyperpolarized transported O depolarized 2 ptsIn terms of function when a presynaptic neuron receiving input from a nearby inhibitory neuron, it can be inferred that the both neurons firing won't be synchronized activation of the inhibitory neuron is nullified activation of the presynaptic neurotransmitter release is enhanced this type of interaction represents a regulatory input of neuronal activation
- A local anesthetic (Na+ channel blocker) should do which of the following Make the resting membrane potential more negative Decrease the after hyperpolarization Block the rising phase of the AP None of theseWhich statement is true regarding chemical neurotransmission? O An IPSP closer to the soma will diminish the effects of an EPSP on a dendrite Chemical neurotransmission is mediated by gap junctions All neurotransmitter receptors are located on the postsynaptic cell membrane Neurotransmitters remain in the synaptic cleft indefinitelyAction potentials propagate in one direction because of: O K+ Voltage-gated Channels become inactive for a while O Nat Voltage-gated Channels become inactive for a while O K+ nongated Channels become inactive for a while O Nat nongated Channels become inactive for a while