Diagram the 5 step pathogenesis cycle for coli O157:H7, an extracellular, intestinal pathogen acquired by consuming contaminated food/water. Be sure to include the role of exoenzymes and the Shiga exotoxin in your diagram. Explain the pathogenesis of Listeria monocytogenes. Be sure to include temperature regulation, intracellular growth, and at risk groups in your discussion.

Diagram the 5 step pathogenesis cycle for coli O157:H7, an extracellular, intestinal pathogen acquired by consuming contaminated food/water. Be sure to include the role of exoenzymes and the Shiga exotoxin in your diagram. Explain the pathogenesis of Listeria monocytogenes. Be sure to include temperature regulation, intracellular growth, and at risk groups in your discussion.

Essentials of Pharmacology for Health Professions

7th Edition

ISBN:9781305441620

Author:WOODROW

Publisher:WOODROW

Chapter12: Skin Medications

Section: Chapter Questions

Problem 9CRQ

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Answer the following questions in complete sentences and paragraphs. Draw the diagram by hand: Diagram the 5 step pathogenesis cycle for coli O157:H7, an extracellular, intestinal pathogen acquired by consuming contaminated food/water. Be sure to include the role of exoenzymes and the Shiga exotoxin in your diagram.
Explain the pathogenesis of Listeria monocytogenes. Be sure to include temperature regulation, intracellular growth, and at risk groups in your discussion.
The large intestine contains bacteria, such as E.coli, that synthesize vitamin K and some B vitamins and absorbed into the bloodstream. Answer the following questions: 1. Describe the type of symbiosis best represented by E.coli and human large intestine. 2. When can E.coli become potentially pathogenic (disease-causing)? Explain briefly.
Based on disease syndromes and characteristics, and also on their effect on certain cell cultures and serological groupings, five virulence groups of Escherichia coli are recognized. Briefly distinguish between these virulence groups. [Base your answer on virulence factors, pattern of bacterial attachment to host cells, effects of attachment to host cells, production of toxins, and invasiveness] What will the appearance of Escherichia coli be on MacConkey Agar red and Eosin Methylene Blue (EMB) agar, respectively.
Explain the role of pylori in the pathogenesis of peptic ulcers. Which populations are most susceptible to peptic ulcers? Is H. pylori normal flora? If so, what causes the species to become pathogenic? Construct at table to compare and contrast the 5 steps of microbial pathogenesis. How does extracellular growth differ from intracellular growth? Diagram the 5 step pathogenesis cycle for coli O157:H7, an extracellular, intestinal pathogen acquired by consuming contaminated food/water. Be sure to include the role of exoenzymes and the Shiga exotoxin in your diagram. (2 points) Explain the pathogenesis of Listeria monocytogenes. Be sure to include temperature regulation, intracellular growth, and at risk groups in your discussion.
What are the virulence factors of Salmonella typhi? Explain each factors in simple terms.
Please choose the answers that correctly describe the virulence factors of Staphylococcus aureus. Check All That Apply Coagulase enzyme causes blood plasma to clot. Staphylokinase enzyme destroys red blood cells. Penicillinase enzyme allows S. aureus to grow in the presence of penicillin. Hyaluronidase enzyme promotes Invasion of host tissues. Lipase enzyme causes a fever in the host.
Staphylococcus aureus has five types of toxins that help out with its virulence. They are enterotoxin, exofoliative toxin, toxic shock syndrome toxin, leukocidin and hemolysins. Please choose 3. Explain the damage they specifically can cause and how this is beneficial to the bacteria.
Which of the following is NOT true of bacterial exotoxins? 1. Important in the pathogenesis of many human diseases 2. Their toxic effect can be systemic, affecting cells distant from the primary site of infection 3. None of the other four answers (All are true of bacterial exotoxins) 4. Different exotoxins may affect different types of cells (e.g., nerves, gastrointestinal mucosa) 5. Some exotoxins have two components, A (active) and B (binding)
Write a 1-2 paragraph case study that accurately depicts the disease caused by Clostridium Botulinum. If your organism is transmitted in a specific location or under certain circumstances be sure your patient has been to those locations or engaged in those behaviors that would lead to transmission Have the appropriate timeline in terms of incubation and length of illness. Cover the important symptoms. You do not have to give all possible symptoms, just the typical one. Provide some important laboratory test results without stating the name of your microorganism. Provide the Epidemiology, Pathogenesis, Clinical Manifestations, Laboratory Tests, Treatment and Prevention.
Some bacteria, protozoa, and viruses that cause foodborne illness are: E.coli, Salmonella Norovirus Staphylococcus aureus Clostridium botulinum Campylobacter Clostridium perfringens Hepatitis A. Giardia (mainly water) Listeriosis Select two (2) of the foodborne illnesses listed above and report the following information for each. Disease name and whether it is a bacteria, protozoa, or virus Type of contamination (infection or intoxication) Infectious pathway Possible sources and foods affected Symptoms Incubation period Treatment Current Statistics, how many affected, where, when etc.
During a recent morbidity report at your local hospital, you noted a 50 year old firefighter recovering from smoke inhalation and severe burns received on the job. While recovering from a skin graft, the patient developed sudden and severe upper right quadrant pain accompanied by fever, nausea and vomiting. An abdominal ultrasound demonstrated Murphys sign along with gas in the gallbladder. Escherichia coli Alcaligenes faecalis Bacillus megaterium Bacillus cereus Citrobacter freundii