5. A new mutation that arose in one copy of gene X in a somatic cell resulted in the formation of a tumor. Which of the following pieces of evidence best describes how the new mutation directly caused the tumor? (A) Protein X normally stimulates cell division, and the mutation created an overactive version of protein X. (B) Protein X normally activates a growth hormone receptor, and the mutation decreased the stability of protein X. (C) Protein X normally prevents passage through the cell cycle, and the mutation created an overactive version of protein X. (D) Protein X normally regulates gene expression, and the mutation created an underactive version of protein X that blocked the cell cycle.

Biology: The Dynamic Science (MindTap Course List)
4th Edition
ISBN:9781305389892
Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan
Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan
Chapter19: Genomes And Proteomes
Section19.3: Determining The Functions Of The Genes In A Genome
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5. A new mutation that arose in one copy of gene X in a somatic cell resulted in the formation of
a tumor. Which of the following pieces of evidence best describes how the new mutation
directly caused the tumor?
(A) Protein X normally stimulates cell division, and the mutation created an overactive
version of protein X.
(B) Protein X normally activates a growth hormone receptor, and the mutation
decreased the stability of protein X.
(C) Protein X normally prevents passage through the cell cycle, and the mutation created an
overactive version of protein X.
(D) Protein X normally regulates gene expression, and the mutation created an
underactive version of protein X that blocked the cell cycle.
Transcribed Image Text:5. A new mutation that arose in one copy of gene X in a somatic cell resulted in the formation of a tumor. Which of the following pieces of evidence best describes how the new mutation directly caused the tumor? (A) Protein X normally stimulates cell division, and the mutation created an overactive version of protein X. (B) Protein X normally activates a growth hormone receptor, and the mutation decreased the stability of protein X. (C) Protein X normally prevents passage through the cell cycle, and the mutation created an overactive version of protein X. (D) Protein X normally regulates gene expression, and the mutation created an underactive version of protein X that blocked the cell cycle.
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